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CYP3A4 inhibition by ketoconazole yielded an IC 50 of 0.83 M in iHLOs, comparable to HepaRG cells (0.42 M) but less potent than the IC 50 (0.056 M) from PHHs, demonstrating their applicability for studying CYP-mediated drug-drug interactions (Fig

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Recent studies reveal that nitrogen metabolism within the TME is not autonomously regulated by tumor cells but is instead jointly shaped by complex metabolic interactions and competition between tumor cells and various stromal cells (such as cancer-associated fibroblasts, CAFs) and immune cells (particularly tumor-associated macrophages, TAMs)(Li and Simon 2020)

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